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'Blooming' in the gut: how dysbiosis might contribute to pathogen evolution. Nat Rev Microbiol. 2013;11(4):277-84.
. Gut inflammation can boost horizontal gene transfer between pathogenic and commensal Enterobacteriaceae. Proc Natl Acad Sci U S A. 2012;109(4):1269-74.
. Host-compound foraging by intestinal microbiota revealed by single-cell stable isotope probing. Proc Natl Acad Sci U S A. 2013;110(12):4720-5.
. Inflammation fuels colicin Ib-dependent competition of Salmonella serovar Typhimurium and E. coli in enterobacterial blooms. PLoS Pathog. 2014;10(1):e1003844.
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Mechanisms controlling pathogen colonization of the gut. Curr Opin Microbiol. 2011;14(1):82-91.
. Microbiota-derived hydrogen fuels Salmonella typhimurium invasion of the gut ecosystem. Cell Host Microbe. 2013;14(6):641-51.
. Reporters for Single-Cell Analysis of Colicin Ib Expression in Salmonella enterica Serovar Typhimurium. PLoS One. 2015;10(12):e0144647.
. The role of microbiota in infectious disease. Trends Microbiol. 2008;16(3):107-14.
. Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota. PLoS Biol. 2007;5(10):2177-89.
. Salmonella transiently reside in luminal neutrophils in the inflamed gut. PLoS One. 2012;7(4):e34812.
. Self-destructive cooperation mediated by phenotypic noise. Nature. 2008;454(7207):987-90.
. A simple screen to identify promoters conferring high levels of phenotypic noise. PLoS Genet. 2008;4(12):e1000307.
. . Temperate phages promote colicin-dependent fitness of Salmonella enterica serovar Typhimurium. Environ Microbiol. 2015.
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